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Showing posts with label Garda whistleblower. Show all posts
Showing posts with label Garda whistleblower. Show all posts

Tuesday, April 26, 2016

Donie's Ireland daily news BLOG update

Garda finds nearly third of people it deals with unhappy with police Irish force

     
The troubled Garda Commissioner Nóirín O’Sullivan.

ALMOST A THIRD OF PEOPLE WHO DEAL WITH THE GARDA ARE NOT HAPPY WITH THE FORCE, ITS OWN RESEARCH HAS FOUND.

A survey, not yet published, ordered by Garda headquarters also revealed younger people and men are more likely to be dissatisfied with their experience of policing.
The findings were mentioned during the new Policing Authority’s first ever public questioning of Garda Commissioner Noirin O’Sullivan on how the force is being run.
The Garda chief asked Gurchand Singh, her head of analysis services, to tell the watchdog about how the public perceive policing.
Mr Singh said a recent study carried out for the force found “about an 85% trust level, a medium to high trust level” in the Garda.
“In terms of our satisfaction, we have a very good satisfaction measure of around 70%,” he added.
Pressed further about the breakdown in the figures, Mr Singh revealed higher satisfaction levels among women who had dealt with gardai compared with men.
“Satisfaction increases with age, younger groups had the lowest levels of satisfaction,” he told the Policing Authority meeting at Royal Hospital Kilmainham in Dublin.
He later disclosed that 64% of people aged between 18 and 24 said they were happy with the Garda.
Judith Gillespie, former deputy chief constable of the PSNI who is a member of the Policing Authority, pointed out “that’s more than third [who are] not satisfied.”
Residents in rural areas are also more likely than those who live in cities to be pleased with their experience of the gardai, according to the study.
Policing Authority chairwoman Josephine Feehily has urged Garda chiefs to publish the findings.
Researchers are also asking the public about their fear of crime and how that impacts upon them.
Furthermore, a “booster survey” has been ordered to find out the feelings of minority communities in particular, who traditionally have had higher levels of fear of crime.
Referring to recent gangland murders in Dublin, Ms O’Sullivan said investigations were progressing well and that organised crime remains a priority.
Since last March, when the new merged Garda Drugs and Organised Crime Bureau was set up, there have been 200 arrests by the specialist unit, she said.
More than 36 million euro worth of drugs were taken off the streets and 36 firearms were seized, along with explosives and ammunition, over the same period, she added.
Quizzed about Garda station closures, Garda Deputy Commissioner Donall O’Cualain said there had been a “fairly trouble free transmission” in those areas.
Beefed-up patrols and Garda “clinics” were put into neighbourhoods where stations shut down over recent years, he said.
Ms O’Sullivan also told the meeting that the Garda was in the final stages of its revamped diversity strategy.
Asked if she was happy that just a quarter of her rank and file are women, she said a recent recruitment campaign was focused particularly on women as well as others under-represented such as gay, bisexual and transgender people.
Some 17,000 applicants are from “all walks of life” and there are early indications that a lot of them are from diverse backgrounds, she added.

Meanwhile:-

Final report into Garda whistleblower allegations of misconduct handed into Justice Minister

   
Garda whistle-blower (above left) Maurice McCabe.

THE COMMISSION OF INVESTIGATION INTO GARDA WHISTLEBLOWER MAURICE MCCABE’S ALLEGATIONS OF MISCONDUCT IN THE FORCE HAS SUBMITTED ITS FINAL REPORT TO JUSTICE MINISTER FRANCES FITZGERALD TODAY.

The report by Justice Kevin O’Higgins was handed into the Department of Justice shortly after 5pm this evening.
The Commission examined allegations made by Garda Sgt McCabe surrounding the conduct of gardai in the Cavan/Monaghan district.
The complaints ranged from the mishandling of murder, assault and abduction investigations by gardai, along with allegations that files were manipulated and altered by members of the force.
Judge O’Higgins also looked at the handling of Sgt McCabe’s complaints by former Justice Minister Alan Shatter and former Garda Commissioner Martin Callinan.
Mr Shatter and Mr Callinan were forced from office in the wake of these allegations and other controversies which engulfed the justice system two years ago.
Mr Shatter has always maintained he acted appropriately throughout the Garda scandals, including his handling of Sgt McCabe’s allegations of malpractice in the Cavan/Monaghan district.
The O’Higgins Commission was established following a recommendation by barrister Sean Guerin who first reviewed Sgt McCabe’s allegations.
Mr Guerin was commission to review Sgt McCabe’s claims by Taoiseach Enda Kenny following political pressure from Fianna Fáil leader Micheál Martin and other Opposition TDs.

Scientists have now solved a major diabetes mystery

WE FINALLY UNDERSTAND HOW THE IMMUNE SYSTEM HELPS CAUSE TYPE 1 DIABETES.

    
A type 1 person giving themselves an injection of insulin.

SCIENTISTS HAVE TAKEN A BIG STEP IN BETTER UNDERSTANDING HOW TYPE 1 DIABETES WREAKS HAVOC ON THE BODY, WHICH COULD LEAD TO NOVEL WAYS TO BOTH TREAT AND PREVENT THE DISEASE.

It was previously known that the chronic autoimmune disease involves the immune system attacking four molecules, called autoantigens, in the pancreas. However, diabetes experts have long speculated that a fifth molecule must also be under attack — but they hadn’t been able to identify it until now.
Researchers in the U.K. and Italy have discovered the fifth and final molecule, called tetraspanin-7. Their work could improve diabetes prediction and treatment, said Dr. Michael Christie, the reader in biomedical sciences at the University of Lincoln in England who led the research.
“The discovery that tetraspanin-7 is a major target of immunity in diabetes now provides us with a complete picture of what the immune system recognizes in individual patients, will assist in identifying individuals at risk through detection of antibodies to the protein and will allow the development of procedures to block the tetraspanin-7 immune response as part of a strategy to prevent the disease,” Christie told The Huffington Post on Monday.
Diabetes is characterized by high levels of sugar in the blood. For people with Type 1 diabetes — about 1.25 million children and adults in the U.S., according to the American Diabetes Association — this is because the body fails to produce any or enough insulin, a hormone that helps to take sugar from the blood to other parts of the body.
The disease is currently treated with insulin injections. Children who develop Type 1 diabetes must inject insulin several times a day for the rest of their lives and constantly monitor their blood glucose. Yet they’re still at risk of experiencing complications affecting their eyes, feet, circulation or nervous system, Christie said.
People with Type 1 diabetes tend to have antibodies in their blood that are specifically linked to each of the molecules that are attacked by the immune system. Tests that identify who might be at risk of developing Type 1 diabetes detect these antibodies — the greater the number of different antibodies found, the higher the risk may be.
The discovery that tetraspanin-7 is a major target of immunity in diabetes now provides us with a complete picture of what the immune system recognizes in individual patients…”Research leader Dr. Michael Christie
For the research, published last month in the journal Diabetes, scientists analyzed blood samples from patients with Type 1 diabetes, and used the antibodies linked to tetraspanin-7 to identify the molecule. They also collected some previous data on the properties of the molecule.
When the researchers were able to bind patients’ antibodies to tetraspanin-7, they knew they had made a groundbreaking discovery.
“We were surprised that we were finally able to discover the identity of the target of antibodies in Type 1 diabetes after such a long period, with many groups worldwide on the hunt for it during this time,” Christie said. “We almost gave up at one stage — our initial test for binding of patients’ antibodies to tetraspanin-7 was negative! — but we then realized that perhaps the test we were using was flawed, so we tried a different approach which worked nicely.”
The researchers concluded that the five major targets of the immune system’s response in Type 1 diabetes are insulin, an enzyme called Glutamate decarboxylase, the proteins IA-2 and Zinc transporter-8, and of course tetraspanin-7.
The more technically named molecules are largely involved in the secreting or storing of insulin, BBC News reported.
The research can be used to better identify people at risk of Type 1 diabetes and later inform the crucial development of therapies, Dr. Emily Burns of Diabetes UK, the charity that co-funded the study with the Society for Endocrinology, said in a statement.
“In order to prevent Type 1 diabetes, we need to fully understand how the immune response that damages insulin-producing cells develops in the first place,” she said. “Dr. Christie’s impressive research is helping us to do just that.”

NUI Galway scientists make breast cancer breakthrough

  

RESEARCHERS FROM NUI GALWAY HAVE MADE A BREAKTHROUGH IN THEIR STUDIES INTO BREAST CANCER TREATMENT, WHICH COULD AFFECT A SIGNIFICANT SEGMENT OF SUFFERERS OF THE DISEASE.

Led by Drs Sanjeev and Ananya Gupta in NUI Galway, the paper published in Nature hones in on a single protein that plays a pivotal role for certain sufferers of breast cancer, those who are ‘oestrogen receptor positive’.
XBP1 is the protein in question, with Sanjeev and his team establishing that it increases the production of NCOA3, which helps the cancer cells avoid anti-oestrogen treatment. Using this information, the suggestion is treatment that uses an XBP1 inhibitor could help oestrogen treatment get the job done.
Oestrogen (and progesterone) are in abundance in women’s bodies, potentially serving as fuel for cancerous cells. Hormone therapy, also called endocrine therapy, adds, blocks, or removes those chemicals to treat the disease.
The NUI Galway team claims one-third of breast cancer patients treated with hormonal therapy suffer a relapse within 15 years, which has proved difficult to understand for scientists. Cancer cells’ reliance on XBP1 as a shield could, therefore, be used against them by allowing tailored inhibitors to pull down the cancer’s defences.
Ananya Gupta said: “The next step is to identify a suitable therapeutic target in the XBP1-NCOA3 pathway. XBP1 is a transcription factor, and transcription factors have been very difficult to target with small molecules. We look forward to developing new ways to target this molecule in breast cancer.”
The project was supported by Breast Cancer Now, with the organisation’s Dr Richard Berks hopeful of potential improvements to anti-hormone treatments.
“We look forward to further research to find out whether blocking this protein could reduce the risk of a patient’s breast cancer spreading or returning, ultimately helping to stop women dying from the disease. It’s crucial that we continue to find ways to make breast cancer therapies even more effective, and match individual patients with the treatments most likely to work for them.”

Dublin Tech Summit aims to attract 10,000 attendees

ORGANISER WANTS TO ENSURE CAPITAL REMAINS FOCAL POINT FOLLOWING WEB SUMMIT DEPARTURE

    

DUBLIN TECHNOLOGY SUMMIT ORGANISER BEN ENGLISH: “WE WANT TO BUILD A POSITIVE INTERNATIONAL MESSAGE OUT OF THE TECH. SUMMIT NEXT FEBRUARY.”

The Web Summit may have departed for Lisbon, but another technology conference wants to ensure Dublin remains a focal point on the global conference map.
The Dublin Tech Summit is aiming to attract more than 10,000 attendees to the Convention Centre next February, reaffirming the capital’s reputation as a strong place for enterprise, technology and innovation.
“We want to build a positive international message out of the tech summit,” conference organiser Ben English said.
Uber’s lead engineer Rafi Krikorian, MongoDB chief marketing officer Meagen Eisenberg and Adblock chief executive Till Faida are among the speakers for the event, which will take place on February 15th and 16th.
Other speakers will include PayPal vice-president of global operations Louise Phelan, Kantar chief executive Eric Salama, Hassle.com co-founder Jules Coleman, Startupboat founder Paula Schwarz, and Kathleen Mitchell, vice-president of Stella & Dot.
The conference will focus on seven themes: business, fintech, the internet of things, medtech, big data, social enterprise and creativity. Organisers are expecting more than 200 speakers and investors.
Mr English said the event would be a more intimate affair than Web Summit. “We’re focused on bridging the gap between start-ups, scale-ups and established companies . . . Attendees are saying they want to build relationships in a more intimate conference venue.”
The summit will invite start-ups, companies, investors and entrepreneurs to a number of events over the course of the next year, with the first on May 27th, when conference organisers will open the London Stock Market.
Mr English said negotiations had begun with Dublin City Council, Enterprise Ireland and the Irish Hotels Federation to avoid problems with wifi and increasing hotel prices, which became contentious issues for Web Summit organisers.

Earth getting greener due to rising carbon dioxide levels, global snapshot shows

    

RISING CARBON DIOXIDE LEVELS IN THE ATMOSPHERE HAVE INCREASED PLANT GROWTH ACROSS THE PLANET OVER THE PAST THREE DECADES, A NEW STUDY HAS NOW FOUND.

SOME KEY POINTS?

  • A few areas on Earth have browned but the vast majority have greened
  • Plant coverage has grown by 18 million square kilometres in a few decades
  • The main cause is increase in atmospheric CO2
  • Nitrogen from agricultural fertilisers has also contributed
The most comprehensive modelling of remote sensing data so far shows the area on Earth covered by plants in this time has increased by 18 million square kilometres — about 2.5 times the size of the Australian continent — largely due to the fertilising effect of carbon dioxide (CO2).
“[The greening] has the ability to fundamentally change the cycling of water and carbon in the climate system,” said Dr Zaichun Zhu, from Peking University in China and lead author of the new study, which appears today in the journal Nature Climate Change.
“This is a snapshot of humans’ global influence on the functioning of the entire global biosphere,” said co-author CSIRO’s Dr Pep Canadell of the Global Carbon Project.
The new findings come from analysis of data from the past 33 years taken by three different satellite missions that measured the green light reflected by photosynthesising leaves.
Dr Canadell said the remote sensing data was run through 10 global environmental change models to identify the cause of the greening.
About 85% of Earth’s ice-free land is covered by plants, and each year photosynthesis soaks up about one quarter of the nearly 10 billion tonnes of carbon emitted by humans.
Drs Zhu, Canadell and colleagues found that the 46 parts per million increase in atmospheric CO2 between 1982 and 2009 was responsible for 50 to 70 per cent of the observed greening.
“Carbon fertilisation is the dominant process for greening across the globe, particularly in the tropics because there’s so much leaf area there,” Dr Canadell said.
Scientists have long established humans as a key source of atmospheric CO2 through the use of chemical signatures that distinguishes carbon from different sources.
“The growth of CO2 in the atmosphere is almost exclusively due to fossil fuel burning and deforestation,” said Dr Canadell.
The new study found other causes of the greening, including nitrogen from agricultural fertilisers.
Surprising finding given increase in world droughts?
Dr Canadell said the greening has surprised scientists who expected to see more browning, given the increase in droughts associated with global warming.
While the researchers found between 25 to 50% of all vegetated areas of the land have become greener, only 4 per cent have become browner.
These included Mongolia, Argentina and areas of North America close to Alaska.
While south-eastern Australia also showed browning, overall the Australian continent was greening, said Dr Canadell.
While a greener Earth might seem like a positive from CO2-induced global warming, along with milder winters and longer growing seasons, he said there were many more negative impacts — including rising sea levels and severe weather.
“These will eventually outweigh by far any benefit from the greening,” he said.    

Friday, May 9, 2014

Donie's Ireland daily news BLOG Update

Mary Lou McDonald accuses Ruairi Quinn of sexism in a heated Dail exchange

 

QUINN REFERS TO MCDONALD’S ‘MOTHERLY CONCERN’ FOR LABOUR PARTY

Sinn Féin deputy leader Mary Lou McDonald has accused the Labour Minister for Education of sexism during heated Dáil exchanges on Alan Shatter’s resignation.
Sinn Féin deputy leader Mary Lou McDonald has accused the Labour Minister for Education of sexism during heated Dáil exchanges on Alan Shatter’s resignation.
Speaking in the Dáil today, Ruairí Quinn said he never ceased to be amazed at the warm affection and “motherly concern’’ Ms McDonald expressed for the well being and future of the Labour party. “I cherish it deeply and know that we are well liked and minded by you…and that if we ever begin to go somewhat astray, you will, in a careful way, look after us,’’ he added.
Ms McDonald said Mr Quinn’s reference to her as motherly, “to the cackles of your almost exclusively male audience’’, did not go down well with her or mothers who might be watching the proceedings. “I do not appreciate the tone of that remark,’’ she added.
She said that she knew “a sexist undertone’’ when she heard it.
Earlier, Ms McDonald said Mr Shatter had accepted no responsibility for what had gone wrong under his remit. “But, then, why should he, when the Taoiseach and the Tánaiste and the Cabinet, and the backbenchers of Fine Gael and Labour, have defended his actions in this Dáil and in the media ?,’’ she added.
She said the House was being told that Tánaiste Eamon Gilmore had known about Mr Shatter’s resignation in mid-afternoon. “He certainly did not know about it at lunchtime and your colleague, Joan Burton, did not know about these matters early yesterday morning because she was on the airwaves, reiterating her confidence in Alan Shatter,’’ she added.
Fianna Fáil leader Micheál Martin said it was surprising that Mr Gilmore was defending Mr Shatter, even at lunchtime yesterday. This was against a background of the Taoiseach’s office receiving the Guerin report on Tuesday evening, he added.
Mr Quinn said all he knew was that the Tánaiste had been informed of the resignation in mid-afternoon yesterday. Only the Tánaiste could give a precise answer, he added.

Irish inflation edges up to 0.3% as threat of deflation lingers

 

LATEST CSO DATA SUGGEST EDUCATION, ALCOHOL AND TOBACCO AMONG THE MAIN DRIVERS

A period of deflation or negative price growth has been identified as a serious risk to Irish economic recovery.
Inflation edged up to 0.3 per cent last month, with higher prices for education, alcohol and tobacco among the main drivers.
However, the latest consumer price index, compiled by theCentral Statistics Office (CSO), suggests inflationary pressure in the economy remains weak.
The CSO figures indicated the average basket of consumer goods and services in April was 0.3 per cent higher than a year earlier, up from the 0.2 per cent rate recorded in March, but still below the euro zone average of 0.7 per cent.
A period of deflation or negative price growth could hamper Ireland’s recovery, as it makes debt more difficult to service.
A breakdown of the CSO’s index showed average education costs rose by 4.5 per cent year on year, mainly as a result of the recent hike in college fees.
Alcohol and cigarette prices were also stronger, rising 3.7 per cent over the past 12 months, reflecting increases in excise duty announced in the last budget.
Prices in the CSO’s miscellaneous goods and services section, which includes a range of items from hairdressing to financial services and insurance, rose by 3.2 per cent year on year.
Restaurant and hotel prices also rose by 2 per cent.
Conversely, the average cost of clothing and footwear fell by 3.7 per cent, while the cost of communications, which covers phone bills, dropped by 3.3 per cent.
The figures also showed prices for household equipment and routine house maintenance fell by 3.1 per cent.
In addition, the price of food and non-alcoholic beverages being sold in shops and supermarkets fell by 2.1 per cent year on year.
The CSO said the divisions which caused the largest downward contribution to prices over the year were food and non-alcoholic beverages; clothing and footwear; and household furnishing and maintenance.
This reflects the relatively weak level of consumer demand on the high street, and possibly discounting by retailers.
Davy stockbrokers said headline inflation has been largely depressed by external factors, namely ECB rates and oil prices.
It noted that energy prices were down 2.2 per cent in the year to April, while mortgage interest fell 11 per cent.
If these factors were excluded inflation would put inflation at 1.3 per cent – the fastest rate of growth since January last year, the brokerage said.
“We expect the inflation rate to gradually pick up in the near term as the economy recovers but to remain below 1 per cent for most of 2014.”
“The bottom line is that demand in the economy is not quite as weak as the headline inflation rate suggests,” it said.
At the moment, the local property tax is not included in the CSO’s consumer price index.

Ming Flanagan claims Gardaí induced people to sell drugs to

‘boost crime figures’

 

New Garda whistleblower ‘taking grave risk’ by going to confidential recipient, The Dáil told. 

Independent TD Luke ‘Ming’ Flanagan has claimed senior Gardaí induced young people to sell drugs to undercover officers in an effort to boost crime detection figures.
In the Dáil this morning he named a new whistleblower, serving Garda Nick Keogh, based in Athlone, whom he said was today giving information to the interim confidential recipient, retired judge Patrick McMahon and was taking a “grave risk” by going public.
The Roscommon-South Leitrim TD said the garda was forced to go public because in his Garda station whistleblowers were being referred to as “snitches”.
Mr Flanagan cited a number of “grave allegations” made by the garda including the cover-up of the original file of an incident, where the file was stolen and the incident removed from the pulse system.
New statements were created and an accused was threatened by a garda to plead guilty to an offence, he said.
According to Mr Flanagan, the garda’s greatest concern was a drugs operation where there was a “systematic and orchestrated effort by high ranking Garda officers to induce and coerce Irish citizens, in this case with no previous criminal convictions, to buy drugs from dealers and in doing so putting them in personal danger and in turn to sell drugs to undercover gardaí without making any profit, thus boosting crime detection figures for arrests”.
Mr Flanagan said a “grave aspect” of the operation, was that a significant and well recognised drug dealer in the area – “long associated with a senior member of the drugs unit” – was not targeted.
He asked Minister for Education Ruairí Quinn, who was taking leaders’ questions: “What are you going to do to reassure this person that he won’t be bullied, he won’t have a rat hung on his door”.
He asked would the Government let him know that he was a “hero” and not a “rat”.
Mr Quinn said the Labour Party and the Government was committed to having a proper Ombudsman for gardaí and a proper complaints procedure and introducing a Garda authority, so that the incidents Mr Flanagan was talking about would not reoccur.
Mr Flanagan is running as a European election candidate in the upcoming elections.

Prostate Cancer the Symptoms, Diagnosis & Treatments

  
Prostate cancer is one of the most common forms of cancer in men: Almost 240,000 men in the United States are diagnosed with prostate cancer each year, according to the National Cancer Institute.
The prostate is a gland located beneath a man’s bladder, toward the front of the rectum. About the size of a walnut, it surrounds the urethra, the tube that carries urine from the bladder. The prostate produces seminal fluid, which nourishes and transports sperm.
Prostate cancer usually grows slowly and is often confined to the prostate gland. When it’s detected early, the chances of successfully treating prostate cancer are greater.

Risks and symptoms

  There are no early warning signs of prostate cancer, according to the Mayo Clinic. In more advanced stages — when the prostate has enlarged — symptoms may include trouble urinating, blood in the urine or semen, erectile dysfunction or pain in the lower back, hips or thighs.
According to the American Cancer Society, about 1 man in 7 will be diagnosed with prostate cancer during his lifetime. The disease is more prevalent in men older than 50 years, and about 6 cases in 10 occur in men older than 65 years.
For reasons that are not well understood, prostate cancer is more common among African-American men and Caribbean men of African ancestry. It’s less common, however, in Asia, Africa, Central and South America than it is in Europe, North America, Australia and other regions.
Researchers have also found that prostate cancer can run in families, suggesting a genetic link. A low-fiber diet that is high in meat and dairy products and workplace exposure to certain toxins have also been linked to a higher risk of prostate cancer.
In 2014, about 29,480 men are expected to die of prostate cancer, though most men diagnosed with the condition do not die from it, according to the American Cancer Society. More than 2.5 million men with prostate cancer are still alive today.
There is no universally agreed-upon schedule or time-frame for screening men who may be at risk for prostate cancer. Some medical organizations recommend screening all men over the age of 50, and earlier screening for men who may be at higher risk, such as African-Americans or men with a family history of prostate cancer.
Men who have a weak urine stream, difficulty stopping a urine stream, a dribbling stream or blood in their urine or semen may wish to talk to their health care provider about those or any related symptoms, regardless of age.
It should be noted that some of the symptoms of prostate cancer — weak or dribbling urine stream — are similar to the symptoms of benign prostatic hyperplasia, or BPH, a common condition that’s sometimes referred to as prostate gland enlargement.
Your doctor or health care provider will probably request a urinalysis (urine test) to rule out an infection or other condition, according to the Mayo Clinic. A digital rectal exam (DRE), in which your doctor will insert a gloved finger into your rectum to check your prostate, is also commonly performed.
A prostate-specific antigen (PSA) test is a blood test that determines the amount of PSA in your bloodstream. PSA is a substance produced by the prostate; elevated PSA levels may be an indication of infection, inflammation, enlargement or cancer.
  If any of these procedures detects an abnormality, your doctor may recommend additional tests, such as an ultrasound of your prostate or a biopsy of tissue collected from your prostate. [5 Things You Should Know About Prostate Cancer]

Treatment

Treatment of prostate cancer depends in part on your age, general health and the degree to which the cancer has spread, or its stage. Stage I prostate cancer is described as a very early cancer that’s non-aggressive and confined to a small area of the prostate.
Stage II prostate cancer is more aggressive and may have spread to both sides of the prostate gland. Stages III and IV describe cancer that has spread beyond the prostate gland to other tissues or organs, according to the National Cancer Institute.
Many doctors recommend active surveillance or “watchful waiting” — including blood tests, rectal exams, biopsies and other procedures — to see if more aggressive treatment is necessary. Surveillance may be an option when prostate cancer is very slow growing or limited in scope.
There are two types of radiation therapy used to treat prostate cancer. External beam radiation uses a large machine to direct radiation to the prostate and other affected tissue. Brachytherapy, in which numerous small implants (about the size of a grain of rice) are placed inside the body, delivers a low dose of radiation over a long period of time.
Prostate cancer cells rely on the male hormone testosterone to grow. Hormone therapy can reduce the amount of testosterone in your body, thereby limiting the growth of cancer cells. Some doctors recommend drugs that stop the body from producing testosterone; other drugs may be used to block the hormone from reaching cancer cells.
Some doctors may also recommend surgery to remove the testicles (which produce testosterone) in a procedure known as an orchiectomy.
 Robotic surgery (Eg. left picture) is a option your doctor and consultant may propose which involves the Urologist to remove the prostate gland, this is known as a (prostatectomy), and this could be done for all stages of prostate cancer.
In most cases, the surgery will involve the remove of the entire prostate gland and nearby lymph nodes. A prostatectomy can result in urinary incontinence and erectile dysfunction.
For advanced cases of prostate cancer, some doctors recommend chemotherapy to kill cancer cells, or immunotherapy, which stimulates the immune system to destroy cancer cells.

Life after prostate cancer treatment

Regular checkups and other procedures are usually scheduled for people who have been treated for prostate cancer. These may include a DRE, a PSA test or other procedures. Your doctor will also be able to offer help in coping with the side effects of prostate cancer treatment.
Prostate cancer can recur following treatment. Your health care provider will address any changes and suggest additional treatments as appropriate.
Men who have had prostate cancer may benefit from cancer support groups, individual counseling, stress management classes, help from social workers or alternative therapies such as relaxation techniques, spiritual guidance, meditation and exercise.

Polar Bears DNA could help cure obesity?

DNA MAY HOLD A SECRET TO BOOSTING METABOLISM AND PREVENTING HEART DISEASE 

  
  • The animal’s DNA is uniquely evolved to cope with a high fat diet
  • They have mutated genes involved in fatty acid metabolism
  • Explains how they eat a high fat diet without suffering clogged arteries
  • This discovery could help scientists find new ways to treat human obesity
Polar bears may hold the answer to the obesity crisis in their genes, new research has shown.
A study of the animal’s DNA revealed they are uniquely evolved to cope with a high fat diet that would prove disastrous to a human.
The bears prey on blubber-rich seals, and also scavenge the fatty carcasses of whales.
Polar bears could hold the key to the obesity crisis in their genes, new research suggests
Half their body weight consists of fat and their cholesterol levels are sky high, yet they are untroubled by heart disease.
Now scientists believe they know the polar bear’s secret – several mutated genes involved in fatty acid metabolism and cardiovascular function.
The genes appear to be crucial to the bears’ adaptation to extreme conditions in the high Arctic and may explain how they avoid clogged arteries.
Scientists hope understanding how they work will help them find new ways to fight human obesity.
Lead researcher Professor Rasmus Nielsen, from the University of California at Berkeley, U.S., said: ‘The promise of comparative genomics is that we learn how other organisms deal with conditions that we also are exposed to.
‘For example, polar bears have adapted genetically to a high fat diet that many people now impose on themselves.
Studying how bears cope with a hugely high fat diet could help scientists tackle obesity in humans
‘If we learn a bit about the genes that allows them to deal with that, perhaps that will give us tools to modulate human physiology down the line.’
Colleague Dr Eline Lorenzen, also from Berkeley, said: ‘For polar bears, profound obesity is a benign state.
‘We wanted to understand how they are able to cope with that.’
The study, published in the journal Cell, showed that the polar bear is a much younger species than was previously thought.
COULD OTHER BEARS ALSO HELP IN THE FIGHT AGAINST OBESITY?
  
In December last year it was revealed that scientists are also studying grizzly bears in the hope they could solve the obesity crisis.
A study is being carried out into the creatures because they are able to eat up to 58,000 calories a day without becoming overweight.
Despite eating such huge amounts, they do not suffer heart attacks or clogged arteries – and they don’t develop diabetes.
As a result, the U.S. drug maker Amgen is studying 12 of the bears at Washington State University in a bid to work out how they do it.
Test results so far suggest that bears modify their sensitivity to the hormone insulin, which controls how sugars and fats are broken down and used for energy.
As they put on weight before hibernation, bears seem to be more sensitive to insulin, Dr Kevin Corbit said.
But when hibernation begins they seem to shut off their responsiveness to the hormone completely.
Dr Corbit plans to spend the next two years working out how exactly the mechanism works.
His work will be assisted by the sequencing of of the bears’ genome which he hopes will be completed soon.
Polar bears diverged from brown bears less than 500,000 years ago, compared with previous estimates of up to five million years.
After setting off on their own evolutionary path they rapidly changed to adapt to Arctic life.
Not only did their fur become white, but genes that affected their metabolism and heart function altered.
One gene, known as APOB, plays a role in moving cholesterol from the bloodstream into cells, thereby reducing the risk of heart disease.
Danish co-author Dr Eske Willerslev, from the University of Copenhagen, said: ‘Such a drastic genetic response to chronically elevated levels of fat and cholesterol in the diet has not previously been reported.
‘It certainly encourages a move beyond the standard model organisms in our search for the underlying genetic causes of human cardiovascular disease.’
The research involved sequencing and analysing the complete genomes, or genetic codes, of 79 polar bears from Greenland and 10 brown bears from around the world.
It comes at a time when the polar bear population – estimated at no more than 20,000 to 25,000 – is declining and the animal’s habitat, Arctic sea ice, rapidly vanishing.
As higher latitudes warm, brown bears are moving further north and occasionally interbreeding with their distant Arctic cousins.
The animals’ ability to interbreed is the result of a genetic relationship a tenth of the distance between humans and chimpanzees, the scientists said.
‘All the unique adaptations polar bears have to the Arctic environment must have evolved in a very short amount of time,’ Professor Nielsen said.
‘These adaptations include not only a change from brown to white fur and development of a sleeker body, but big physiological and metabolic changes as well.
‘There has been a lot of debate about it, but I think we really nailed down what the divergence time is between them, and it is surprisingly recent.’

Birds can’t use their migratory direction process because our devices make to much noise

 

Wavelength interference from electronics and AM radio stations disrupt the internal magnetic compasses of European migratory birds, explains a report published yesterday in Nature.

Seven years of double-blind experiments (new researchers replicating old experiments) led the report’s co-author,Henrik Mouritsen, of the University of Oldenburg, Germany, to conclude that cities disrupt migratory bird patterns.
The Earth’s magnetic field, virtually mapped for the first time earlier this week, clues in European robins to their migratory direction, the report details. But when electromagnetic waves from our devices pollute the atmosphere, the birds don’t know which way to fly.
Mouritsen covered wooden huts housing his robins with aluminum plating (the traditional method of blinding birds from magnetic clues such as the sun or stars, Naturewrites). When his team grounded the plating—reducing the electromagnetic interference on campus from 50 kilohertz to five megahertz—they found that the birds could orient themselves. Ungrounded, however, the frequencies were so strong that the bird could not find their way.
Until Mouritsen moved his experiment from a rural field site into the college town, there was very little evidence of humans’ electromagnetic radiation affecting birds’ migratory patterns.
He poses this question to Nature: “If birds can’t use one of their most significant compasses when they are in towns, what effect will that have on survival?”